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Let me share some of them with you. There are several theories including free radical damage , an inability to use glucose properly, vitamin deficiencies or environmental toxins. This illness affects a third of people over the age of 85 in the U. Hippocrates knew the importance of food in healing the body back in B.

Modern science is catching up. Plaque is characterized by deposits of a toxic protein called beta-amyloid in the spaces between nerve cells in the brain. Think of tangles of knotted threads of the tau protein found within brain cells. The new study used PET imaging to study the brain for changes and is the first to demonstrate how lifestyle factors directly influence abnormal proteins in people with subtle memory loss who have not yet been diagnosed with dementia.

Keep in mind, though, that we need more research on how HIIT impacts the brain. However, a previous study did find that steady state cardio creates more brain neurons compared to weight training or HIIT. For now, just focus on any physical activity and getting into a healthy BMI range. They found that those who worked closely with people in complex work situations were able to tolerate brain damage better than those who worked in more isolated settings.

In the lab, the plant compounds blocked the disease by easing cellular inflammation and removing toxic amyloid proteins on brain cells.

Alternative Medicine and Alzheimer's Disease

Clinical trials are now needed to see if the promising results hold true in humans, too. Taking anticholinergic drugs also led to worse scores on memory tests. Certain antidepressants, COPD and asthma medications, along with drugs for overactive bladder issues, could also fall in the anticholinergic category. Therefore, if you need these medications, talk to your doctor or pharmacist to see if safer alternatives exist. What people read on the Internet is all about these two proteins but that's not a problem. People have a hard time moving for what they think.

If you look at dementia and causes of Alzheimer's and so on if I Google it right now it's going to talk to you about the two proteins are causes of Alzheimer's. The causes are disrupting genetics. So it starts developing at a younger age.

Effects of medicinal plants on Alzheimer's disease and memory deficits

But what's the reason behind Alzheimer's or dementia. Classical homeopathy aims to find or match one remedy to the whole condition the person is suffering. Symptoms presented - such as difficulties in thinking, forgetfulness, reasoning, communication as well as feeling lost in familiar places and unable to recognize one's family members and friends with lots of words and loss of judgment - is only the beginning; taken alone, these symptoms do not define the choice of the correct homeopathic remedy. Baryta Carb can help those who are regressing into a fearful behavior, timidity with shyness and lack confidence.

There is the loss of memory and some patients may suffer from chronic glandular disorders. Natrum Sulf can often bring relief to those who dwell on past hurts and unpleasant events, possibly the loss of a partner. They may feel sad and lonely, these people often have arthritic painful joints as well. Nux Vomica patients are often angry, quarrelsome and insensitive to other's feelings; however, they themselves are extremely sensitive to everything and tend to abuse alcohol.

They are often very constipated with small black pellets. They are claimed to enhance memory and rejuvenate cognitive functions [ 21 — 23 ]. Several Ayurvedic medicines have been exploited for the treatment and management of acute and chronic neurological diseases. These formulations induce specific effects on brain functions, such as increase in blood flow and maintenance of memory [ 11 ]. Ashwagandha Withania somnifera , fam.

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Solanaceae , or Indian Ginseng, is a common herb used in Ayurvedic medicine as an adaptogen or antistress agent. Ashwagandha root contains a large variety of compounds including 12 alkaloids, 40 withanolides, and several sitoindosides and flavonoids [ 24 — 26 ]. Withaferin A WL-A and withanolide A are two constituents which show similar pharmacokinetic profiles, except that the oral bioavailability for WL-A is 1.

These components produce antistress, antioxidant, and immunomodulatory effects in acute models of experimental stress [ 28 — 30 ]. According to Ayurvedic medicine, Ashwagandha constituents provide a number of healthful effects such as youthful state of physical and mental health and increase in happiness. It is not only given to children as tonics but is consumed by the middle-aged and elderly to increase longevity [ 31 , 32 ].

It is suggested that WL-A activates the translocation of Nrf2 to the nucleus, where the transcription factor upregulates the expression of neuroprotective proteins, such as heme oxygenase-1 [ 34 , 35 ]. Treatment of human neuroblastoma SK-N-SH cells with methanolic extracts of Ashwagandha root results in dendrite extension, neurite outgrowth, and synapse formation [ 36 , 37 ].

WL-A also attenuates the expression of semaphorin 3A to facilitate neural regeneration. The beneficial effects of Ashwagandha root constituents in neurodegenerative diseases may be due to their neurite promoting, antioxidant, anti-inflammatory, antiapoptotic, and anxiolytic activities, as well as their ability to improve mitochondrial dysfunction and restore energy levels and increase levels of antioxidant defenses such as reduced glutathione [ 38 ] Figure 3.

Based on the above information, it is proposed that WL-A is an important candidate for the treatment of dementia and neurodegenerative diseases, since it is able to repair damaged neuronal networks [ 33 , 38 ]. Ashwagandha is a safe herb [ 39 , 40 ], although a few people experience diarrhea or nausea after consuming the root.

Alternative Medicine and Alzheimer's Disease

It should not be taken with barbiturate-type sedatives, since the herb can increase the effectiveness of these drugs. Ashwagandha can cross the blood-brain barrier and lower inflammation in the brain. The half-lives of Ashwagandha in the circulation and the brain are not known.

Large multicenter clinical trials of Ashwagandha in patients with dementia have not been performed. It is derived from the rhizome of Curcuma longa , which belongs to the family Zingiberaceae. It has antioxidant, anti-inflammatory, and cancer chemopreventive properties [ 42 ]. Curcumin reduces oxidative damage and improves cognitive functions related to the aging process. It induces antioxidant effects by modulating the Nrf2-keap1 pathway and reduces genomic instability events [ 43 ]. Nrf2 is primarily present in the cytoplasm, where it is bound with the Kelch-like ECH-associated protein 1 Keap1.

Interaction of curcumin with Keap 1 releases Nrf2, which migrates into the nucleus and binds as a heterodimer to antioxidant responsive elements in DNA, to initiate target gene expression. Nrf2-regulated genes include antioxidant enzymes, molecular chaperones, DNA repair enzymes, and anti-inflammatory response proteins [ 44 ] Figure 4. These proteins promote the reduction in ROS generation while increasing the ability of the cell to repair any subsequent damage [ 44 , 45 ].

Curcumin attenuates neuroinflammation through the inhibition of phospholipase A 2 PLA 2 and cyclooxygenase COX-2 enzymes associated with the metabolism of neural membrane phospholipids to prostaglandins Figure 4. The absorption rate and bioavailability of curcumin can be increased by consuming it with black pepper Piper nigrum.

Studies have indicated that piperine, an active ingredient in black pepper, increases the bioavailability and bioefficacy of curcumin by inhibiting its glucuronidation [ 49 ]. Interestingly, consumption of piperine and curcumin has been found to protect against chronic unpredictable stress-induced cognitive impairment and oxidative damage in mice [ 50 , 51 ].

The half-lives of curcumin in the circulation and the brain are not known. Large multicenter clinical trials of curcumin in patients with dementia have not been performed, although a number of small studies have been conducted in healthy individuals. Bacopa monnieri belongs to the family Scrophulariaceae and is found throughout the Indian subcontinent in wet, damp, and marshy areas [ 54 ]. It has many branches with small oblong leaves and purple flowers. This plant is not only used for the treatment of a number of nervous system disorders such as insomnia, anxiety, and epilepsy, but also used for enhancing memory and the intellect [ 55 ].

In Ayurvedic medicine, Bacopa monnieri is used as a memory enhancing, anti-inflammatory, analgesic, antipyretic, sedative, and antiepileptic agent, which acts as a nootropic repairing damaged neurons and improving brain function. Bacosides inhibit lipoxygenase activity and scavenge free radicals. They protect neural cells of the prefrontal cortex, hippocampus, and striatum against cytotoxicity and DNA damage implicated in AD.

Alzheimer's disease drug shows early promise

Bacosides increase glutathione peroxidase, chelate iron [ 58 , 59 ], and enhance nitric oxide-mediated cerebral vasodilation, leading to improvements in total memory score [ 59 ]. This leads to an increase in protein and RNA turnover in certain brain regions such as the hippocampus [ 57 ]. Furthermore, the combination of bacosides A and B not only induces antistress effects [ 61 ], but also protects the brain against smoking-induced membrane damage [ 62 ] and D -galactosamine-induced liver injury [ 63 ].

While there are no studies that prove that Bacopa monnieri causes side effects, it has been observed that excessive intake of Bacopa monnieri may lead to stomach upset, diarrhea, and nausea. The intake and use of Bacopa monnieri should be avoided by pregnant and breastfeeding women.

The half-lives of bacosides in the circulation and the brain are not known. A number of clinical trials have been carried out in human subjects on the use of Bacopa monnieri for improving cognition. Bacopa monnieri treatment for 3 months decreases the rate of forgetting of newly acquired information in human subjects between 40 and 65 years of age [ 55 ].

The roots and rhizomes of Acorus calamus are used in Ayurvedic medicine on a regular basis for the treatment of insomnia, melancholia, neurosis, loss of memory, and remittent fevers. Convolvulus pluricaulis has been shown to improve learning and memory in rodents [ 68 ]. Saussurea lappa has been reported to produce anti-inflammatory activity [ 69 ].

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Old clarified butter or old ghee is described in Ayurveda as a memory enhancer, anticonvulsant, and anti-inflammatory agent [ 70 , 71 ].